Kinase-Independent Functions for Itk in TCR-Induced Regulation of Vav and the Actin Cytoskeleton-Reference-Cited by-同舟云学术

Kinase-Independent Functions for Itk in TCR-Induced Regulation of Vav and the Actin Cytoskeleton

Published:2005-01-20 Issue:3 Volume:174 Page:1385-1392
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Dombroski Derek,Houghtling Richard A.,Labno Christine M.,Precht Patricia,Takesono Aya,Caplen Natasha J.,Billadeau Daniel D.,Wange Ronald L.,Burkhardt Janis K.,Schwartzberg Pamela L.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference46 articles.

1. The regulation of actin remodeling during T-cell-APC conjugate formation

2. The Rho-family GTP exchange factor Vav is a critical transducer of T cell receptor signals to the calcium, ERK, and NF- B pathways

3. Vav is a regulator of cytoskeletal reorganization mediated by the T-cell receptor

4. Wiskott-Aldrich Syndrome Protein-Deficient Mice Reveal a Role for WASP in T but Not B Cell Activation

5. Antigen Receptor–Induced Activation and Cytoskeletal Rearrangement Are Impaired in Wiskott-Aldrich Syndrome Protein–Deficient Lymphocytes

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