Despite Increased Type 1 IFN, Autoimmune Nonobese Diabetic Mice Display Impaired Dendritic Cell Response to CpG and Decreased Nuclear Localization of IFN-Activated STAT1
Author:
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Reference59 articles.
1. Innate immunity and the pathogenesis of type 1 diabetes
2. The importance of the Non Obese Diabetic (NOD) mouse model in autoimmune diabetes
3. Type 1 Diabetes: Etiology, Immunology, and Therapeutic Strategies
4. A comprehensive guide to antibody and T-cell responses in type 1 diabetes
5. Checkpoints in the progression of autoimmune disease: lessons from diabetes models.
Cited by 9 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Innate immune stimulation of whole blood reveals IFN-1 hyper-responsiveness in type 1 diabetes;Diabetologia;2020-06-05
2. CpG‐ODN‐mediated TLR9 innate immune signalling and calcium dyshomeostasis converge on the NFκB inhibitory protein IκBβ to drive IL1α and IL1β expression;Immunology;2020-03-18
3. Dendritic Cells in Autoimmune Disease;The Autoimmune Diseases;2020
4. Type-I Interferons Inhibit Interleukin-10 Signaling and Favor Type 1 Diabetes Development in Nonobese Diabetic Mice;Frontiers in Immunology;2018-07-16
5. Restoration of the type I IFN–IL-1 balance through targeted blockade of PTGER4 inhibits autoimmunity in NOD mice;JCI Insight;2018-02-08
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