Cyclin-Dependent Kinase Inhibitor Cdkn2c Deficiency Promotes B1a Cell Expansion and Autoimmunity in a Mouse Model of Lupus-Reference-Cited by-同舟云学术

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Cyclin-Dependent Kinase Inhibitor Cdkn2c Deficiency Promotes B1a Cell Expansion and Autoimmunity in a Mouse Model of Lupus

Published:2012-08-15 Issue:6 Volume:189 Page:2931-2940
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Potula Hari-Hara S. K.,Xu Zhiwei,Zeumer Leilani,Sang Allison,Croker Byron P.,Morel Laurence

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference42 articles.

1. Origins and Functions of B-1 Cells with Notes on the Role of CD5

2. Cell Cycle Control Mechanisms in B-1 and B-2 Lymphoid Subsets

3. Cyclin D2 is essential for BCR-mediated proliferation and CD5 B cell development

4. Disruption of Cyclin D3 Blocks Proliferation of Normal B-1a Cells, but Loss of Cyclin D3 Is Compensated by Cyclin D2 in Cyclin D3-Deficient Mice

5. Early Induction of Cyclin D2 Expression in Phorbol Ester–responsive B-1 Lymphocytes

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