CCR3 Is Required for Tissue Eosinophilia and Larval Cytotoxicity After Infection withTrichinella spiralis-Reference-Cited by-同舟云学术

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CCR3 Is Required for Tissue Eosinophilia and Larval Cytotoxicity After Infection withTrichinella spiralis

Published:2002-06-01 Issue:11 Volume:168 Page:5730-5736
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Gurish Michael F.,Humbles Alison,Tao Hong,Finkelstein Stella,Boyce Joshua A.,Gerard Craig,Friend Daniel S.,Austen K. Frank

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference50 articles.

1. The Biology of Chemokines and their Receptors

2. Molecular cloning and characterization of a human eotaxin receptor expressed selectively on eosinophils.

3. Chemokine-induced eosinophil recruitment. Evidence of a role for endogenous eotaxin in an in vivo allergy model in mouse skin.

4. Chemokine receptor usage by human eosinophils. The importance of CCR3 demonstrated using an antagonistic monoclonal antibody.

5. The C-C chemokine receptor CCR3 participates in stimulation of eosinophil arrest on inflammatory endothelium in shear flow.

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