IL-11 Induces Encephalitogenic Th17 Cells in Multiple Sclerosis and Experimental Autoimmune Encephalomyelitis-Reference-Cited by-同舟云学术

IL-11 Induces Encephalitogenic Th17 Cells in Multiple Sclerosis and Experimental Autoimmune Encephalomyelitis

Published:2019-07-24 Issue:5 Volume:203 Page:1142-1150
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Zhang Xin,Kiapour Nazanin^ORCID,Kapoor Sahil^ORCID,Khan Tabish,Thamilarasan Madhan,Tao Yazhong,Cohen Stephanie,Miller Ryan^ORCID,Sobel Raymond A.^ORCID,Markovic-Plese Silva

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference34 articles.

1. Intramuscular Interferon Beta-1A Therapy Initiated during a First Demyelinating Event in Multiple Sclerosis

2. IL-11 Induces Th17 Cell Responses in Patients with Early Relapsing-Remitting Multiple Sclerosis

3. C-C chemokine receptor 6–regulated entry of TH-17 cells into the CNS through the choroid plexus is required for the initiation of EAE

4. Interleukins 1β and 6 but not transforming growth factor-β are essential for the differentiation of interleukin 17–producing human T helper cells

5. Activation of the signal transducer gp130 by interleukin-11 and interleukin-6 is mediated by similar molecular interactions

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