Activation of NF-κB in Virus-Infected Macrophages Is Dependent on Mitochondrial Oxidative Stress and Intracellular Calcium: Downstream Involvement of the Kinases TGF-β-Activated Kinase 1, Mitogen-Activated Kinase/Extracellular Signal-Regulated Kinase Kinase 1, and IκB Kinase-Reference-Cited by-同舟云学术

Activation of NF-κB in Virus-Infected Macrophages Is Dependent on Mitochondrial Oxidative Stress and Intracellular Calcium: Downstream Involvement of the Kinases TGF-β-Activated Kinase 1, Mitogen-Activated Kinase/Extracellular Signal-Regulated Kinase Kinase 1, and IκB Kinase

Published:2003-06-15 Issue:12 Volume:170 Page:6224-6233
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Mogensen Trine H.,Melchjorsen Jesper,Höllsberg Per,Paludan Søren R.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference49 articles.

1. Requirements for the Induction of Interleukin-6 by Herpes Simplex Virus-Infected Leukocytes

2. Expression of TNF-α by Herpes Simplex Virus-Infected Macrophages Is Regulated by a Dual Mechanism: Transcriptional Regulation by NF-κB and Activating Transcription Factor 2/Jun and Translational Regulation Through the AU-Rich Region of the 3′ Untranslated Region

3. Herpes Simplex Virus Selectively Induces Expression of the CC Chemokine RANTES/CCL5 in Macrophages through a Mechanism Dependent on PKR and ICP0

4. Molecular Pathways in Virus-Induced Cytokine Production

5. NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune Responses

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