Functional analysis of the sheep Wilson disease protein (sATP7B) in CHO cells
Author:
Publisher
Elsevier BV
Subject
Cell Biology,General Medicine,Histology,Pathology and Forensic Medicine
Reference43 articles.
1. TGN38 is maintained in the trans-Golgi network by a tyrosine-containing motif in the cytoplasmic domain;Bos;EMBO. J.,1993
2. Null mutation of the murine ATP7B (Wilson disease) gene results in intracellular copper accumulation and late-onset hepatic nodular transformation;Buiakova;Hum. Mol. Genet.,1999
3. The Wilson disease gene is a putative copper transporting P-type ATPase similar to the Menkes gene;Bull;Nature Genet.,1993
4. Gene amplification of the Menkes (MNK; ATP7A) P-type ATPase gene of CHO cells is associated with copper resistance and enhanced copper efflux;Camakaris;Hum. Mol. Genet.,1995
5. Retrieval of TGN proteins from the cell surface requires endosomal acidification;Chapman;EMBO J.,1994
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