In LNCaP Cells Inhibition of BCL-2 by Antisense Oligonucleonucleotides Results in Compensatory Changes in Apoptosis
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Peertechz Publications Private Limited
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https://www.peertechzpublications.com/articles/SJGGT-1-101.pdf
Reference14 articles.
1. 1. Yamanaka K, Miyake H, Zangemeister-wittke U, Jansen B, Gleave M (2004) Novel bispecific antisense oligonucleotides inhibiting both BCL-2 and Bcl-xL expression induce apoptosis and enhance chemosensitivity in human androgen-independent prostate cancer cells. Proceedings AACR 45.
2. Insulin-like growth factor-binding protein-5 (IGFBP5) supports prostate cancer cell proliferation;Takahara;J Urol,2009
3. Bispecific antisense oligonucleotides with multiple binding sites for the treatment of prostate tumors and their applicability to combination therapy;Rubenstein;Meth Find Clin Pharmacol,2006
4. Combination chemotherapy employing bispecific antisense oligonucleotides having binding sites directed against an autocrine regulated growth pathway and BCL-2 for the treatment of prostate cancer;Rubenstein;Med Oncol,2007
5. Multigene targeting of signal transduction pathways for the treatment of breast and prostate tumors. Comparison between combination therapies employing bispecific oligonucleotides with either Rapamycin or Paclitaxel;Rubenstein;Med Oncol,2009
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