APE1 recruits ATRIP to ssDNA in an RPA-dependent and -independent manner to promote the ATR DNA damage response
Author:
Affiliation:
1. Department of Biological Sciences, University of North Carolina at Charlotte
2. School of Data Science, University of North Carolina at Charlotte
3. Center for Biomedical Engineering and Science, University of North Carolina at Charlotte
Abstract
Funder
National Institutes of Health
University of North Carolina at Charlotte
Publisher
eLife Sciences Publications, Ltd
Subject
General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,General Medicine,General Neuroscience
Link
https://cdn.elifesciences.org/articles/82324/elife-82324-v1.pdf
Reference75 articles.
1. Human AP Endonuclease 1 (Ape1): From mechanistic insights to Druggable target in cancer;Abbotts;Cancer Treatment Reviews,2010
2. Direct binding to Replicationprotein A (RPA)-Coated single-stranded DNA allows recruitment of the ATR activator Topbp1 to sites of DNA damage;Acevedo;The Journal of Biological Chemistry,2016
3. Small-molecule inhibitors of Ape1 DNA repair function: An overview;Al-Safi;Current Molecular Pharmacology,2012
4. ATRIP binding to replication protein A-single-stranded DNA promotes ATR-ATRIP localization but is Dispensable for Chk1 Phosphorylation;Ball;Molecular Biology of the Cell,2005
5. Checking on DNA damage in S phase;Bartek;Nature Reviews. Molecular Cell Biology,2004
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