AT&sub1; Receptor Blockade Prevents Microvascular Dysfunction Induced by Ischemia/Reperfusion Injury
Author:
Affiliation:
1. Department of Internal Medicine I, School of Medicine, Oita University
2. Department of Laboratory Medicine, School of Medicine, Oita University
Publisher
Japan Atherosclerosis Society
Subject
Biochemistry, medical,Cardiology and Cardiovascular Medicine,Internal Medicine
Link
https://www.jstage.jst.go.jp/article/jat/13/5/13_5_231/_pdf
Reference33 articles.
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2. 2) Carden DL and Granger DN: Pathophysiology of ischemia-reperfusion injury. J Pathol, 2000; 190: 255-266
3. 3) Hahn AW, Jonas U, Buhler FR, and Resink TJ: Activation of human peripheral monocytes by angiotensin II, FEBS Lett, 1994; 347: 178-180
4. 4) Krejcy K, Eichler HG, Jilma B, Kapiotis S, Wolzt M, Zanaschka G, Gasic S, Schutz W, and Wagner O: Influence of angiotensin II on circulating adhesion molecules and blood leukocyte count in vivo. Can J Physiol Pharmacol, 1996; 74: 9-14
5. 5) Kim JA, Berliner JA, and Nadler JL: Angiotensin II increases monocyte binding to endothelial cells. Biochem Biophys Res Commun, 1996; 226: 862-868
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