AICAR Attenuates TNFα-Induced Inappropriate Secretion of Monocyte Chemoattractant Protein-1 and Adiponectin in 3T3-L1 Adipocytes
Author:
Affiliation:
1. Department of Pediatrics, Showa University School of Medicine
2. Showa University Graduate School of Health Sciences
Publisher
Japan Atherosclerosis Society
Subject
Biochemistry, medical,Cardiology and Cardiovascular Medicine,Internal Medicine
Link
https://www.jstage.jst.go.jp/article/jat/23/12/23_34835/_pdf
Reference33 articles.
1. 1) Gustafson B: Adipose tissue, inflammation and atherosclerosis. J Atheroscler Thromb. 2010; 30: 332-341
2. 2) Funahashi T and Matsuzawa Y: Adiponectin and the cardiometabolic syndrome: an epidemiological perspective. Best Pract Res Clin Endocrinol Metab. 2014; 28: 93-106
3. 3) Yamauchi T and Kadowaki T: Adiponectin receptor as a key player in healthy longevity and obesity-related diseases. Cell Metab. 2013; 17: 185-196
4. 4) Araki S, Dobashi K, Kubo K, and Asayama K, Shirahata A: High molecular weight, rather than total, adiponectin levels better reflect metabolic abnormalities associated with childhood obesity. J Clin Endocrinol Metab. 2006; 91: 5113-5116
5. 5) Bijland S, Mancini SJ, and Salt IP: Role of AMP-activated protein kinase in adipose tissue metabolism and inflammation. Clin Sci. 2013; 124: 491-507
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1. Salicylate Sodium Suppresses Monocyte Chemoattractant Protein-1 Production by Directly Inhibiting Phosphodiesterase 3B in TNF-α-Stimulated Adipocytes;International Journal of Molecular Sciences;2022-12-24
2. Mesenchymal Stem Cell Differentiation into Adipocytes Is Equally Induced by Insulin and Proinsulin In Vitro;International Journal of Stem Cells;2017-11-30
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