Angiotensin Ⅱ Activates MCP-1 and Induces Cardiac Hypertrophy and Dysfunction via Toll-like Receptor 4-Reference-Cited by-同舟云学术

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Angiotensin Ⅱ Activates MCP-1 and Induces Cardiac Hypertrophy and Dysfunction via Toll-like Receptor 4

Published:2015 Issue:8 Volume:22 Page:833-844
ISSN:1340-3478
Container-title:Journal of Atherosclerosis and Thrombosis
language:en
Short-container-title:JAT

Author:

Matsuda Susumu¹,Umemoto Seiji²,Yoshimura Koichi³,Itoh Shinichi¹,Murata Tomoaki⁴,Fukai Tohru⁵,Matsuzaki Masunori¹

Affiliation:

1. Department of Medicine and Clinical Science, Yamaguchi University Graduate School of Medicine

2. Center for Clinical Research, Yamaguchi University Hospital

3. Department of Surgery and Clinical Science, Yamaguchi University Graduate School of Medicine

4. Institute of Experimental Animals, Science Research Center, Yamaguchi University

5. Departments of Medicine (Section of Cardiology) and Pharmacology, Center for Cardiovascular Research, University of Illinois at Chicago

Publisher

Japan Atherosclerosis Society

Subject

Biochemistry, medical,Cardiology and Cardiovascular Medicine,Internal Medicine

Link

https://www.jstage.jst.go.jp/article/jat/22/8/22_27292/_pdf

Reference44 articles.

1. 1) Kurdi M, Booz GW: New take on the role of angiotensin II in cardiac hypertrophy and fibrosis. Hypertension, 2011; 57: 1034-1038

2. 2) Touyz RM, Briones AM: Reactive oxygen species and vascular biology: implications in human hypertension. Hypertens Res, 2011; 34: 5-14

3. 3) Chinnaiyan KM, Alexander D, McCullough PA: Role of angiotensin II in the evolution of diastolic heart failure. J Clin Hypertens (Greenwich), 2005; 7: 740-747

4. 4) Kono M, Kisanuki A, Ueya N, Kubota K, Kuwahara E, Takasaki K, Yuasa T, Mizukami N, Miyata M, Tei C: Left ventricular global systolic dysfunction has a significant role in the development of diastolic heart failure in patients with systemic hypertension. Hypertens Res, 2010; 33: 1167-1173

5. 5) Lalande S, Johnson BD: Diastolic dysfunction: a link between hypertension and heart failure. Drugs Today (Barc), 2008; 44: 503-513

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