Mechanisms of WNK1 and WNK4 interaction in the regulation of thiazide-sensitive NaCl cotransport
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Reference24 articles.
1. Human Hypertension Caused by Mutations in WNK Kinases
2. WNK1, a Novel Mammalian Serine/Threonine Protein Kinase Lacking the Catalytic Lysine in Subdomain II
3. Wnk1 kinase deficiency lowers blood pressure in mice: A gene-trap screen to identify potential targets for therapeutic intervention
4. WNK kinases regulate thiazide-sensitive Na-Cl cotransport
5. Molecular pathogenesis of inherited hypertension with hyperkalemia: The Na-Cl cotransporter is inhibited by wild-type but not mutant WNK4
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1. Navigating the multifaceted intricacies of the Na+-Cl− cotransporter, a highly regulated key effector in the control of hydromineral homeostasis;Physiological Reviews;2024-07-01
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3. Expression of the kidney anion exchanger 1 affects WNK4 and SPAK phosphorylation and results in claudin-4 phosphorylation;Heliyon;2023-11
4. Thirty years of the NaCl cotransporter: from cloning to physiology and structure;American Journal of Physiology-Renal Physiology;2023-10-01
5. Kidney and blood pressure regulation—latest evidence for molecular mechanisms;Clinical Kidney Journal;2023-01-24
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