JAK2-V617F promotes venous thrombosis through β1/β2 integrin activation-Reference-Cited by-同舟云学术

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JAK2-V617F promotes venous thrombosis through β1/β2 integrin activation

Published:2018-07-19 Issue:10 Volume:128 Page:4359-4371
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Edelmann Bärbel,Gupta Nibedita,Schnoeder Tina M.,Oelschlegel Anja M.,Shahzad Khurrum,Goldschmidt Jürgen^ORCID,Philipsen Lars^ORCID,Weinert Soenke,Ghosh Aniket,Saalfeld Felix C.,Nimmagadda Subbaiah Chary,Müller Peter,Braun-Dullaeus Rüdiger,Mohr Juliane,Wolleschak Denise,Kliche Stefanie,Amthauer Holger,Heidel Florian H.^ORCID,Schraven Burkhart^ORCID,Isermann Berend,Müller Andreas J.,Fischer Thomas

Funder

German Ministry of Education and Research

Else-Kröner-Fresenius Foundation

German Research Council

Jose-Carreras-Foundation

Thuringian Ministry for Research

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/90312/files/pdf

Reference76 articles.

1. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera

2. A Gain-of-Function Mutation ofJAK2in Myeloproliferative Disorders

3. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis

4. JAK Inhibition with Ruxolitinib versus Best Available Therapy for Myelofibrosis

5. Safety and Efficacy of INCB018424, a JAK1 and JAK2 Inhibitor, in Myelofibrosis

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4. Exacerbation of thromboinflammation by JAK2 V617F mutation worsens the prognosis of cerebral venous sinus thrombosis;Blood Advances;2024-06-25

5. Neutrophil-specific expression of JAK2-V617F or CALRmut induces distinct inflammatory profiles in myeloproliferative neoplasia;Journal of Hematology & Oncology;2024-06-09

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