Hepatic glucose sensing is required to preserve β cell glucose competence-Reference-Cited by-同舟云学术

Hepatic glucose sensing is required to preserve β cell glucose competence

Published:2013-03-15 Issue:4 Volume:123 Page:1662-1676
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Seyer Pascal,Vallois David,Poitry-Yamate Carole,Schütz Frédéric,Metref Salima,Tarussio David,Maechler Pierre,Staels Bart,Lanz Bernard,Grueter Rolf,Decaris Julie,Turner Scott,da Costa Anabela,Preitner Frédéric,Minehira Kaori,Foretz Marc,Thorens Bernard

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference60 articles.

1. Contribution of de novo fatty acid synthesis to hepatic steatosis and insulin resistance: lessons from genetically engineered mice

2. Glucose has to be phosphorylated to activate glycogen synthase, but not to inactivate glycogen phosphorylase in hepatocytes

3. New perspectives in the regulation of hepatic glycolytic and lipogenic genes by insulin and glucose: a role for the transcription factor sterol regulatory element binding protein-1c

4. Normal hepatic glucose production in the absence of GLUT2 reveals an alternative pathway for glucose release from hepatocytes

5. Liver Hyperplasia and Paradoxical Regulation of Glycogen Metabolism and Glucose-sensitive Gene Expression in GLUT2-null Hepatocytes

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