Calpain activation impairs neuromuscular transmission in a mouse model of the slow-channel myasthenic syndrome-Reference-Cited by-同舟云学术

Calpain activation impairs neuromuscular transmission in a mouse model of the slow-channel myasthenic syndrome

Published:2007-10-01 Issue:10 Volume:117 Page:2903-2912
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Groshong Jason S.,Spencer Melissa J.,Bhattacharyya Bula J.,Kudryashova Elena,Vohra Bhupinder P.S.,Zayas Roberto,Wollmann Robert L.,Miller Richard J.,Gomez Christopher M.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference50 articles.

1. A newly recognized congenital myasthenic syndrome attributed to a prolonged open time of the acetylcholine-induced ion channel

2. THE SLOW CHANNEL SYNDROME

3. A ?-subunit mutation in the acetylcholine receptor channel gate causes severe slow-channel syndrome

4. Slow-Channel Myasthenic Syndrome Caused By Enhanced Activation, Desensitization, and Agonist Binding Affinity Attributable to Mutation in the M2 Domain of the Acetylcholine Receptor α Subunit

5. New mutations in acetylcholine receptor subunit genes reveal heterogeneity in the slow-channel congenital myasthenic syndrome

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