FAVL elevation in human tumors disrupts Fanconi anemia pathway signaling and promotes genomic instability and tumor growth-Reference-Cited by-同舟云学术

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FAVL elevation in human tumors disrupts Fanconi anemia pathway signaling and promotes genomic instability and tumor growth

Published:2010-05-03 Issue:5 Volume:120 Page:1524-1534
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Zhang Jun,Zhao Deping,Park Hwan Ki,Wang Hong,Dyer Roy B.,Liu Wanguo,Klee George G.,McNiven Mark A.,Tindall Donald J.,Molina Julian R.,Fei Peiwen

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference41 articles.

1. Fanconi's Anaemia in the Genetics of Neoplasia

2. Fanconi Anemia

3. Cancer in Fanconi anemia

4. Biallelic Inactivation of BRCA2 in Fanconi Anemia

5. BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ

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2. TFG-maintaining stability of overlooked FANCD2 confers early DNA-damage response;Aging;2020-10-24

3. A new look at molecular biology of breast cancer;Cancer Biology & Therapy;2018-09-06

4. DNA methylation at the vicinity of the proximal polyadenylation site in FANCD2 gene involves human malignancy;Cell Cycle;2018-09-02

5. Fanconi Anemia complementation group C protein in metabolic disorders;Aging;2018-06-21

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