Disrupted ER membrane protein complex–mediated topogenesis drives congenital neural crest defects-Reference-Cited by-同舟云学术

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Disrupted ER membrane protein complex–mediated topogenesis drives congenital neural crest defects

Published:2020-01-06 Issue:2 Volume:130 Page:813-826
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Marquez Jonathan^ORCID,Criscione June,Charney Rebekah M.,Prasad Maneeshi S.,Hwang Woong Y.,Mis Emily K.,García-Castro Martín I.,Khokha Mustafa K.

Funder

National Institute of General Medical Sciences

National Institute of Dental and Craniofacial Research

Hartwell Foundation

National Institute of Child Health and Human Development

Edward Mallinckrodt, Jr. Foundation

Paul and Daisy Soros Fellowship for New Americans

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/129308/files/pdf

Reference85 articles.

1. Comprehensive Characterization of Genes Required for Protein Folding in the Endoplasmic Reticulum

2. The ubiquitous and ancient ER membrane protein complex (EMC): tether or not?;Wideman;F1000Res,2015

3. EMC Is Required to Initiate Accurate Membrane Protein Topogenesis

4. dPob/EMC is essential for biosynthesis of rhodopsin and other multi-pass membrane proteins in Drosophila photoreceptors;Satoh;Elife,2015

5. Biosynthesis of ionotropic acetylcholine receptors requires the evolutionarily conserved ER membrane complex

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