Genetically programmed alternative splicing of NEMO mediates an autoinflammatory disease phenotype
Author:
Funder
Indiana University School of Medicine
NIAMS
NIAID
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/128808/files/pdf
Reference57 articles.
1. The NEMO adaptor bridges the nuclear factor-κB and interferon regulatory factor signaling pathways
2. NF-kappa B activation by a signaling complex containing TRAF2, TANK and TBK1, a novel IKK-related kinase
3. Novel cross-talk within the IKK family controls innate immunity
4. A Novel IRAK1–IKKε Signaling Axis Limits the Activation of TAK1–IKKβ Downstream of TLR3
5. NEMO is a key component of NF-κB– and IRF-3–dependent TLR3-mediated immunity to herpes simplex virus
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