USP25 inhibition ameliorates Alzheimer’s pathology through the regulation of APP processing and Aβ generation
Author:
Funder
CIHR
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/152170/files/pdf
Reference42 articles.
1. BACE2, as a novel APP θ‐secretase, is not responsible for the pathogenesis of Alzheimer's disease in Down syndrome
2. Increased BACE1 maturation contributes to the pathogenesis of Alzheimer's disease in Down syndrome
3. BACE2, a conditional β-secretase, contributes to Alzheimer’s disease pathogenesis
4. Trisomy of human chromosome 21 enhances amyloid-β deposition independently of an extra copy of APP
5. Mechanistic Analysis of Age-Related Clinical Manifestations in Down Syndrome
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