Brugada syndrome trafficking–defective Nav1.5 channels can trap cardiac Kir2.1/2.2 channels
Author:
Funder
Ministry of economy and competitiveness
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/96291/files/pdf
Reference42 articles.
1. Na+channel function, regulation, structure, trafficking and sequestration
2. The cardiac sodium channel gene SCN5A and its gene product NaV1.5: Role in physiology and pathophysiology
3. Cardiac strong inward rectifier potassium channels
4. The effect of the cardiac membrane potential on the rapid availability of the sodium-carrying system
5. Dynamic reciprocity of sodium and potassium channel expression in a macromolecular complex controls cardiac excitability and arrhythmia
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