Hepatic HIF2 is a key determinant of manganese excess and polycythemia in SLC30A10 deficiency-Reference-Cited by-同舟云学术

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Hepatic HIF2 is a key determinant of manganese excess and polycythemia in SLC30A10 deficiency

Published:2024-04-23 Issue:10 Volume:9 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Prajapati Milankumar^ORCID,Zhang Jared Z.,Chiu Lauren,Chong Grace S.,Mercadante Courtney J.^ORCID,Kowalski Heather L.,Delaney Bradley,Anderson Jessica A.,Guo Shuling,Aghajan Mariam,Bartnikas Thomas B.

Funder

National Institutes of Health

Publisher

American Society for Clinical Investigation

Link

https://insight.jci.org/articles/view/169738/files/pdf

Reference55 articles.

1. Manganese metabolism in humans

2. Genetic Disorders of Manganese Metabolism

3. SLC30A10 Is a Cell Surface-Localized Manganese Efflux Transporter, and Parkinsonism-Causing Mutations Block Its Intracellular Trafficking and Efflux Activity

4. SLC30A10 transporter in the digestive system regulates brain manganese under basal conditions while brain SLC30A10 protects against neurotoxicity

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Manganese transporter SLC30A10 and iron transporters SLC40A1 and SLC11A2 impact dietary manganese absorption;2024-07-20

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