Inhibition of profibrotic microRNA-21 affects platelets and their releasate-Reference-Cited by-同舟云学术

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Inhibition of profibrotic microRNA-21 affects platelets and their releasate

Published:2018-11-02 Issue:21 Volume:3 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Barwari Temo^ORCID,Eminaga Seda,Mayr Ursula,Lu Ruifang,Armstrong Paul C.^ORCID,Chan Melissa V.^ORCID,Sahraei Mahnaz,Fernández-Fuertes Marta,Moreau Thomas,Barallobre-Barreiro Javier,Lynch Marc,Yin Xiaoke^ORCID,Schulte Christian^ORCID,Baig Ferheen,Pechlaner Raimund,Langley Sarah R.,Zampetaki Anna,Santer Peter,Weger Martin,Plasenzotti Roberto^ORCID,Schosserer Markus^ORCID,Grillari Johannes,Kiechl Stefan,Willeit Johann,Shah Ajay M.,Ghevaert Cedric,Warner Timothy D.^ORCID,Fernández-Hernando Carlos,Suárez Yajaira,Mayr Manuel^ORCID

Funder

British Heart Foundation

Deutsche Forschungsgemeinschaft

Austrian Science Fund

National Institutes of Health

Österreichische Forschungsförderungsgesellschaft

American Heart Association

Fondation Leducq

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/123335/files/pdf

Reference59 articles.

1. Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

2. MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

3. MicroRNA expression in response to murine myocardial infarction: miR-21 regulates fibroblast metalloprotease-2 via phosphatase and tensin homologue

4. Role of miR-21 in the pathogenesis of atrial fibrosis

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