Tubular CPT1A deletion minimally affects aging and chronic kidney injury-Reference-Cited by-同舟云学术

Tubular CPT1A deletion minimally affects aging and chronic kidney injury

Published:2024-02-22 Issue:6 Volume:9 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Hammoud Safaa,Ivanova Alla,Osaki Yosuke,Funk Steven,Yang Haichun^ORCID,Viquez Olga,Delgado Rachel,Lu Dongliang,Phillips Mignemi Melanie,Tonello Jane,Colon Selene,Lantier Louise^ORCID,Wasserman David H.^ORCID,Humphreys Benjamin D.^ORCID,Koenitzer Jeffrey^ORCID,Kern Justin,de Caestecker Mark^ORCID,Finkel Toren,Fogo Agnes,Messias Nidia,Lodhi Irfan J.,Gewin Leslie S.^ORCID

Funder

National Institute of Health

U.S. Department of Veterans Affairs

Publisher

American Society for Clinical Investigation

Link

https://insight.jci.org/articles/view/171961/files/pdf

Reference59 articles.

1. Defective fatty acid oxidation in renal tubular epithelial cells has a key role in kidney fibrosis development

2. Stimulation of carnitine palmitoyltransferase 1 improves renal function and attenuates tissue damage after ischemia/reperfusion

3. Renal tubule Cpt1a overexpression protects from kidney fibrosis by restoring mitochondrial homeostasis

4. Lipid Accumulation and Chronic Kidney Disease

5. Structural and Functional Changes With the Aging Kidney

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