DDR1 contributes to kidney inflammation and fibrosis by promoting the phosphorylation of BCR and STAT3-Reference-Cited by-同舟云学术

DDR1 contributes to kidney inflammation and fibrosis by promoting the phosphorylation of BCR and STAT3

Published:2022-02-08 Issue:3 Volume:7 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Borza Corina M.,Bolas Gema,Bock Fabian,Zhang Xiuqi,Akabogu Favour C.,Zhang Ming-Zhi,de Caestecker Mark^ORCID,Yang Min,Yang Haichun,Lee Ethan,Gewin Leslie^ORCID,Fogo Agnes B.,McDonald W. Hayes,Zent Roy^ORCID,Pozzi Ambra^ORCID

Funder

NIH and VA

NIH

Veterans Administration

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/150887/files/pdf

Reference40 articles.

1. Increased risk of death and de novo chronic kidney disease following reversible acute kidney injury

2. Progression of chronic kidney disease: too much cellular talk causes damage

3. Selective pharmacological inhibition of DDR1 prevents experimentally-induced glomerulonephritis in prevention and therapeutic regime

4. DDR1 role in fibrosis and its pharmacological targeting

5. Cell receptor-basement membrane interactions in health and disease: a kidney-centric view;Borza;Curr Top Membr,2015

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