IL-13 promotes functional recovery after myocardial infarction via direct signaling to macrophages
Author:
Funder
NIH
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/172702/files/pdf
Reference54 articles.
1. The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions
2. Monocytes/macrophages prevent healing defects and left ventricular thrombus formation after myocardial infarction
3. Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction
4. An acute immune response underlies the benefit of cardiac stem cell therapy
5. The human heart contains distinct macrophage subsets with divergent origins and functions
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