HDAC9 complex inhibition improves smooth muscle–dependent stenotic vascular disease-Reference-Cited by-同舟云学术

HDAC9 complex inhibition improves smooth muscle–dependent stenotic vascular disease

Published:2019-01-24 Issue:2 Volume:4 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Lino Cardenas Christian L.,Kessinger Chase W.,Chou Elizabeth,Ghoshhajra Brian,Yeri Ashish S.,Das Saumya,Weintraub Neal L.^ORCID,Malhotra Rajeev,Jaffer Farouc A.,Lindsay Mark E.

Funder

NIH

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/124706/files/pdf

Reference27 articles.

1. Smooth muscle phenotypic modulation is an early event in aortic aneurysms

2. Epigenetic Control of Smooth Muscle Cell Differentiation and Phenotypic Switching in Vascular Development and Disease

3. Mutations in smooth muscle α-actin (ACTA2) lead to thoracic aortic aneurysms and dissections

4. MYH11 mutations result in a distinct vascular pathology driven by insulin-like growth factor 1 and angiotensin II

5. Genetic Basis of Thoracic Aortic Aneurysms and Dissections: Focus on Smooth Muscle Cell Contractile Dysfunction

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