TRPM7 kinase is required for insulin production and compensatory islet responses during obesity-Reference-Cited by-同舟云学术

TRPM7 kinase is required for insulin production and compensatory islet responses during obesity

Published:2023-02-08 Issue:3 Volume:8 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Khajavi Noushafarin,Beck Andreas,Riçku Klea,Beyerle Philipp,Jacob Katharina,Syamsul Sabrina F.,Belkacemi Anouar,Reinach Peter S.,Schreier Pascale C.F.,Salah Houssein,Popp Tanja^ORCID,Novikoff Aaron^ORCID,Breit Andreas,Chubanov Vladimir^ORCID,Müller Timo D.^ORCID,Zierler Susanna^ORCID,Gudermann Thomas^ORCID

Funder

German Research Foundation, DFG

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/163397/files/pdf

Reference65 articles.

1. Islet cell failure in type 2 diabetes

2. Regulation of Vertebrate Cellular Mg2+ Homeostasis by TRPM7

3. TRPM7 is the central gatekeeper of intestinal mineral absorption essential for postnatal survival

4. Hypomagnesemia in Patients with Type 2 Diabetes

5. Oral Magnesium Supplementation Improves Insulin Sensitivity and Metabolic Control in Type 2 Diabetic Subjects

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3. Chronic Mg2+ Deficiency Does Not Impair Insulin Secretion in Mice;Cells;2023-07-05

4. Inactivation of TRPM7 Kinase Targets AKT Signaling and Cyclooxygenase-2 Expression in Human CML Cells;Function;2023