Complement receptor C3aR1 controls neutrophil mobilization following spinal cord injury through physiological antagonism of CXCR2
Author:
Funder
The National Health and Medical Research Council
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/98254/files/pdf
Reference65 articles.
1. Neutrophil mobilization and clearance in the bone marrow
2. Neutrophils as protagonists and targets in chronic inflammation
3. Neutrophil recruitment and function in health and inflammation
4. The receptor for complement component C3a mediates protection from intestinal ischemia-reperfusion injuries by inhibiting neutrophil mobilization
5. Is the Complement Activation Product C3a a Proinflammatory Molecule? Re-evaluating the Evidence and the Myth
Cited by 64 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Complement-dependent neuroinflammation in spinal cord injury: from pathology to therapeutic implications;Neural Regeneration Research;2024-06-03
2. Spatiotemporally resolved transcriptome atlas of developing mouse placenta reveals mechanisms of embryonic lethality;2024-05-30
3. Crepuscular rays — The bright side of complement after tissue injury;European Journal of Immunology;2024-05-25
4. Identification of pivotal genes and regulatory networks associated with atherosclerotic carotid artery stenosis based on comprehensive bioinformatics analysis and machine learning;Frontiers in Pharmacology;2024-04-17
5. Neutrophil-inflicted vasculature damage suppresses immune-mediated optic nerve regeneration;Cell Reports;2024-03
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3