Activation-induced cytidine deaminase deficiency accelerates autoimmune diabetes in NOD mice-Reference-Cited by-同舟云学术

Activation-induced cytidine deaminase deficiency accelerates autoimmune diabetes in NOD mice

Published:2018-01-11 Issue:1 Volume:3 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Tan Qiyuan,Tai Ningwen,Li Yangyang,Pearson James^ORCID,Pennetti Sean,Zhou Zhiguang,Wong F. Susan^ORCID,Wen Li

Funder

JDRF

NIH, ADA, Chinese Student and Scholar Council, Shanghai Pujiang Program

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/95882/files/pdf

Reference41 articles.

1. Cellular and molecular pathogenesis of type 1A diabetes

2. B lymphocytes are critical antigen-presenting cells for the initiation of T cell-mediated autoimmune diabetes in nonobese diabetic mice;Serreze;J Immunol,1998

3. Investigation of the Role of B-Cells in Type 1 Diabetes in the NOD Mouse

4. Treatment with CD20-specific antibody prevents and reverses autoimmune diabetes in mice

5. B Lymphocyte Depletion by CD20 Monoclonal Antibody Prevents Diabetes in Nonobese Diabetic Mice despite Isotype-Specific Differences in FcγR Effector Functions

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