Implicating bites from a leishmaniasis sand fly vector in the loss of tolerance in pemphigus
Author:
Funder
National Institutes of Health (NIH)/NIAID
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/123861/files/pdf
Reference28 articles.
1. Advances in pemphigus and its endemic pemphigus foliaceus (Fogo Selvagem) phenotype: A paradigm of human autoimmunity
2. Anti-Desmoglein-1 Antibodies in Onchocerciasis, Leishmaniasis and Chagas Disease Suggest a Possible Etiological Link to Fogo Selvagem
3. Anti-desmoglein-1 antibodies are prevalent in Tunisian patients with hydatidosis and leishmaniasis
4. [Geography of major parasitosis in Tunisia];Ben Rachid;Arch Inst Pasteur Tunis,1984
5. Exposure to Phlebotomus papatasi and/or Leishmania major: Possible Etiologic Link to Tunisian Pemphigus
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1. PpSP32, the Phlebotomus papatasi immunodominant salivary protein, exerts immunomodulatory effects on human monocytes, macrophages, and lymphocytes;Parasites & Vectors;2023-01-02
2. PpSP32, Phlebotomus papatasi immunodominant salivary protein, exerts immunomodulatory effects on human monocytes, macrophages and lymphocytes;2022-11-03
3. From Insect Bites to a Skin Autoimmune Disease: A Conceivable Pathway to Endemic Pemphigus Foliaceus;Frontiers in Immunology;2022-05-27
4. Some Good and Some Bad: Sand Fly Salivary Proteins in the Control of Leishmaniasis and in Autoimmunity;Frontiers in Cellular and Infection Microbiology;2022-02-25
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