SOCS1 is a negative regulator of metabolic reprogramming during sepsis-Reference-Cited by-同舟云学术

SOCS1 is a negative regulator of metabolic reprogramming during sepsis

Published:2017-07-06 Issue:13 Volume:2 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Piñeros Alvarez Annie Rocio^ORCID,Glosson-Byers Nicole,Brandt Stephanie,Wang Soujuan,Wong Hector^ORCID,Sturgeon Sarah,McCarthy Brian Paul,Territo Paul R.,Alves-Filho Jose Carlos,Serezani C. Henrique

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/92530/files/pdf

Reference87 articles.

1. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)

2. Sepsis and septic shock;Hotchkiss;Nat Rev Dis Primers,2016

3. Disease Tolerance as a Defense Strategy

4. Stress, Inflammation, and Defense of Homeostasis

5. Role of Tissue Protection in Lethal Respiratory Viral-Bacterial Coinfection

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