Modulation of the effects of Class-Ib antiarrhythmics on cardiac NaV1.5-encoded channels by accessory NaVβ subunits
Author:
Funder
American Heart Association
National Heart, Lung, and Blood Institute
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
http://insight.jci.org/articles/view/143092/files/pdf
Cited by 11 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Decreased ability to manage increases in reactive oxygen species may underlie susceptibility to arrhythmias in mice lacking Scn1b;American Journal of Physiology-Heart and Circulatory Physiology;2024-10-01
2. The dispensability of 14-3-3 proteins for the regulation of human cardiac sodium channel Nav1.5;PLOS ONE;2024-03-07
3. Biophysical properties of NaV1.5 channels from atrial-like and ventricular-like cardiomyocytes derived from human induced pluripotent stem cells;Scientific Reports;2023-11-24
4. Differential regulation of cardiac sodium channels by intracellular fibroblast growth factors;Journal of General Physiology;2023-03-21
5. Cardiac sodium channel complexes and arrhythmia: structural and functional roles of the β1 and β3 subunits;The Journal of Physiology;2022-12-03
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