CF airway smooth muscle transcriptome reveals a role for PYK2-Reference-Cited by-同舟云学术

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CF airway smooth muscle transcriptome reveals a role for PYK2

Published:2017-09-07 Issue:17 Volume:2 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Cook Daniel P.,Adam Ryan J.,Zarei Keyan,Deonovic Benjamin,Stroik Mallory R.,Gansemer Nicholas D.,Meyerholz David K.,Au Kin Fai,Stoltz David A.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/95332/files/pdf

Reference57 articles.

1. Risk of asthma in heterozygous carriers for cystic fibrosis: A meta-analysis

2. Acute administration of ivacaftor to people with cystic fibrosis and a G551D-CFTR mutation reveals smooth muscle abnormalities;Adam;JCI Insight,2016

3. Regulation of the Cystic Fibrosis Transmembrane Conductance Regulator Channel by β-Adrenergic Agonists and Vasoactive Intestinal Peptide in Rat Smooth Muscle Cells and Its Role in Vasorelaxation

4. Disruption of CFTR chloride channel alters mechanical properties and cAMP-dependent Cl- transport of mouse aortic smooth muscle cells;Robert;J Physiol (Lond),2005

5. Evidence that CFTR is expressed in rat tracheal smooth muscle cells and contributes to bronchodilation;Vandebrouck;Respir Res,2006

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Swine models in translational research and medicine;Veterinary Pathology;2024-01-10

2. CFTR dysfunction in smooth muscle drives TGFβ dependent airway hyperreactivity;Respiratory Research;2023-08-11

3. Subacute TGFβ Exposure Drives Airway Hyperresponsiveness in Cystic Fibrosis Mice through the PI3K Pathway;American Journal of Respiratory Cell and Molecular Biology;2020-05

4. Is PI3K a Villain in Cystic Fibrosis?;American Journal of Respiratory Cell and Molecular Biology;2020-05

5. Subacute TGFβ expression drives inflammation, goblet cell hyperplasia, and pulmonary function abnormalities in mice with effects dependent on CFTR function;American Journal of Physiology-Lung Cellular and Molecular Physiology;2018-09-01

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