Diverting T helper cell trafficking through increased plasticity attenuates autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

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Diverting T helper cell trafficking through increased plasticity attenuates autoimmune encephalomyelitis

Published:2013-12-09 Issue:1 Volume:124 Page:174-187
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Califano Danielle,Sweeney Keith J.,Le Hung,VanValkenburgh Jeffrey,Yager Eric,O’Connor William,Kennedy Jeffrey S.,Jones David M.,Avram Dorina

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference56 articles.

1. A Novel Transcription Factor, T-bet, Directs Th1 Lineage Commitment

2. The Transcription Factor GATA-3 Is Necessary and Sufficient for Th2 Cytokine Gene Expression in CD4 T Cells

3. Mechanisms Underlying Lineage Commitment and Plasticity of Helper CD4 + T Cells

4. The Orphan Nuclear Receptor RORγt Directs the Differentiation Program of Proinflammatory IL-17+ T Helper Cells

5. RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation

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