Tyrosine kinase inhibitor NVP-BGJ398 functionally improves FGFR3-related dwarfism in mouse model-Reference-Cited by-同舟云学术

Tyrosine kinase inhibitor NVP-BGJ398 functionally improves FGFR3-related dwarfism in mouse model

Published:2016-04-11 Issue:5 Volume:126 Page:1871-1884
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Komla-Ebri Davide,Dambroise Emilie,Kramer Ina,Benoist-Lasselin Catherine,Kaci Nabil,Le Gall Cindy,Martin Ludovic,Busca Patricia,Barbault Florent,Graus-Porta Diana,Munnich Arnold,Kneissel Michaela,Di Rocco Federico,Biosse-Duplan Martin,Legeai-Mallet Laurence

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference52 articles.

1. Achondroplasia

2. Achondroplasia

3. Mutations in the gene encoding fibroblast growth factor receptor-3 in achondroplasia

4. Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia

5. Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3

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