Sustained ERK inhibition maximizes responses of BrafV600E thyroid cancers to radioiodine-Reference-Cited by-同舟云学术

Sustained ERK inhibition maximizes responses of BrafV600E thyroid cancers to radioiodine

Published:2016-09-26 Issue:11 Volume:126 Page:4119-4124
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Nagarajah James,Le Mina,Knauf Jeffrey A.,Ferrandino Giuseppe,Montero-Conde Cristina,Pillarsetty Nagavarakishore,Bolaender Alexander,Irwin Christopher,Krishnamoorthy Gnana Prakasam,Saqcena Mahesh,Larson Steven M.,Ho Alan L.,Seshan Venkatraman,Ishii Nobuya,Carrasco Nancy,Rosen Neal,Weber Wolfgang A.,Fagin James A.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference24 articles.

1. Long-Term Outcome of 444 Patients with Distant Metastases from Papillary and Follicular Thyroid Carcinoma: Benefits and Limits of Radioiodine Therapy

2. Integrated Genomic Characterization of Papillary Thyroid Carcinoma

3. Multiple Ras Downstream Pathways Mediate Functional Repression of the Homeobox Gene Product TTF-1

4. RET/PTC-induced dedifferentiation of thyroid cells is mediated through Y1062 signaling through SHC-RAS-MAP kinase

5. Conditional BRAFV600E Expression Induces DNA Synthesis, Apoptosis, Dedifferentiation, and Chromosomal Instability in Thyroid PCCL3 Cells

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