Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer-Reference-Cited by-同舟云学术

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer

Published:2014-04-01 Issue:5 Volume:124 Page:2172-2187
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Li Cunxi,Ma Haiting,Wang Yang,Cao Zheng,Graves-Deal Ramona,Powell Anne E.,Starchenko Alina,Ayers Gregory D.,Washington Mary Kay,Kamath Vidya,Desai Keyur,Gerdes Michael J.,Solnica-Krezel Lila,Coffey Robert J.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

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1. Subtractive hybridization reveals the expression of immunoglobulinlike transcript 7, Eph-B1, granzyme B, and 3 novel transcripts in human plasmacytoid dendritic cells

2. Plac8 and Plac9, novel placental-enriched genes identified through microarray analysis

3. Impaired Host Defense in Mice Lacking ONZIN

4. ONZIN deficiency attenuates contact hypersensitivity responses in mice

5. Plac8 Is an Inducer of C/EBPβ Required for Brown Fat Differentiation, Thermoregulation, and Control of Body Weight

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