c-Cbl–deficient mice have reduced adiposity, higher energy expenditure, and improved peripheral insulin action-Reference-Cited by-同舟云学术

c-Cbl–deficient mice have reduced adiposity, higher energy expenditure, and improved peripheral insulin action

Published:2004-11-01 Issue:9 Volume:114 Page:1326-1333
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Molero Juan C.,Jensen Thomas E.,Withers Phil C.,Couzens Michelle,Herzog Herbert,Thien Christine B.F.,Langdon Wallace Y.,Walder Ken,Murphy Maria A.,Bowtell David D.L.,James David E.,Cooney Gregory J.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference39 articles.

1. Cbl: many adaptations to regulate protein tyrosine kinases

2. Insulin Signaling in Microdomains of the Plasma Membrane

3. APS Facilitates c-Cbl Tyrosine Phosphorylation and GLUT4 Translocation in Response to Insulin in 3T3-L1 Adipocytes

4. CAP defines a second signalling pathway required for insulin-stimulated glucose transport

5. Insulin-stimulated GLUT4 translocation requires the CAP-dependent activation of TC10

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