The natriuretic peptide/guanylyl cyclase–A system functions as a stress-responsive regulator of angiogenesis in mice-Reference-Cited by-同舟云学术

The natriuretic peptide/guanylyl cyclase–A system functions as a stress-responsive regulator of angiogenesis in mice

Published:2009-07-01 Issue:7 Volume:119 Page:2019-2030
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Kuhn Michaela,Völker Katharina,Schwarz Kristine,Carbajo-Lozoya Javier,Flögel Ulrich,Jacoby Christoph,Stypmann Jörg,van Eickels Martin,Gambaryan Stepan,Hartmann Michael,Werner Matthias,Wieland Thomas,Schrader Jürgen,Baba Hideo A.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference47 articles.

1. Structure, Regulation, and Function of Mammalian Membrane Guanylyl Cyclase Receptors, With a Focus on Guanylyl Cyclase-A

2. Genetic Decreases in Atrial Natriuretic Peptide and Salt-Sensitive Hypertension

3. Salt-resistant hypertension in mice lacking the guanylyl cyclase-A receptor for atrial natriuretic peptide

4. Hypervolemic hypertension in mice with systemic inactivation of the (floxed) guanylyl cyclase-A gene by ?MHC-Cre-mediated recombination

5. Cardiac fibrosis in mice lacking brain natriuretic peptide

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