Emerging roles for macrophages in cardiac injury: cytoprotection, repair, and regeneration
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Reference24 articles.
1. Embryonic and Adult-Derived Resident Cardiac Macrophages Are Maintained through Distinct Mechanisms at Steady State and during Inflammation
2. Differential Contribution of Monocytes to Heart Macrophages in Steady-State and After Myocardial Infarction
3. The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an ‘M2’ phenotype in response to Th2 immunologic challenge
4. CCL2/Monocyte Chemoattractant Protein-1 Regulates Inflammatory Responses Critical to Healing Myocardial Infarcts
5. Ly-6C high Monocytes Depend on Nr4a1 to Balance Both Inflammatory and Reparative Phases in the Infarcted Myocardium
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2. Mechanically induced pyroptosis enhances cardiosphere oxidative stress resistance and metabolism for myocardial infarction therapy;Nature Communications;2023-10-02
3. hUC-MSC-EV-miR-24 enhances the protective effect of dexmedetomidine preconditioning against myocardial ischemia–reperfusion injury through the KEAP1/Nrf2/HO-1 signaling;Drug Delivery and Translational Research;2023-08-04
4. Mechanically induced pyroptosis enhanced cardiosphere oxidative stress resistance and metabolism for myocardial infarction therapy;2023-02-28
5. Renewal of embryonic and neonatal-derived cardiac-resident macrophages in response to environmental cues abrogated their potential to promote cardiomyocyte proliferation via Jagged-1–Notch1;Acta Pharmaceutica Sinica B;2023-01
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