TGF-β signaling in myeloproliferative neoplasms contributes to myelofibrosis without disrupting the hematopoietic niche-Reference-Cited by-同舟云学术

TGF-β signaling in myeloproliferative neoplasms contributes to myelofibrosis without disrupting the hematopoietic niche

Published:2022-06-01 Issue:11 Volume:132 Page:
ISSN:1558-8238
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Yao Juo-Chin^ORCID,Oetjen Karolyn A.^ORCID,Wang Tianjiao^ORCID,Xu Haoliang,Abou-Ezzi Grazia,Krambs Joseph R.^ORCID,Uttarwar Salil,Duncavage Eric J.^ORCID,Link Daniel C.

Funder

NIH NHLBI

NIH NCI

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/154092/files/pdf

Reference55 articles.

1. Haematopoietic stem cells and early lymphoid progenitors occupy distinct bone marrow niches

2. CXCL12 in early mesenchymal progenitors is required for haematopoietic stem-cell maintenance

3. Endothelial and perivascular cells maintain haematopoietic stem cells

4. Concise Review: The Malignant Hematopoietic Stem Cell Niche

5. Classification and diagnosis of myeloproliferative neoplasms: The 2008 World Health Organization criteria and point-of-care diagnostic algorithms

Cited by 16 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. CXCR4-enriched T regulatory cells preferentially home to bone marrow and resolve inflammation;iScience;2024-09

2. The CALR mutations enhance the expression of the immunosuppressive proteins GARP and LAP on peripheral blood lymphocytes through increased binding of activated platelets;British Journal of Haematology;2024-08-20

3. The Variation in the Traits Ameliorated by Inhibitors of JAK1/2, TGF-β, P-Selectin, and CXCR1/CXCR2 in the Gata1low Model Suggests That Myelofibrosis Should Be Treated by These Drugs in Combination;International Journal of Molecular Sciences;2024-07-13

4. Overlapping Stromal Alterations in Myeloid and Lymphoid Neoplasms;Cancers;2024-05-30

5. CCN2/CTGF expression does not correlate with fibrosis in myeloproliferative neoplasms, consistent with noncanonical TGF-β signaling driving myelofibrosis;Virchows Archiv;2024-04-11