Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Cited by 76 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
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2. Microfluidic study of retention and elimination of abnormal red blood cells by human spleen with implications for sickle cell disease;Proceedings of the National Academy of Sciences;2023-02-02
3. Genetic and structural basis of the human anti-α-galactosyl antibody response;Proceedings of the National Academy of Sciences;2022-07-08
4. Anti-Gal and Autoimmunity;The Natural Anti-Gal Antibody As Foe Turned Friend In Medicine;2018
5. Why Do We Produce Anti-Gal;The Natural Anti-Gal Antibody As Foe Turned Friend In Medicine;2018
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