Altered trafficking and stability of polycystins underlie polycystic kidney disease-Reference-Cited by-同舟云学术

Altered trafficking and stability of polycystins underlie polycystic kidney disease

Published:2014-11-03 Issue:12 Volume:124 Page:5129-5144
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Cai Yiqiang,Fedeles Sorin V.,Dong Ke,Anyatonwu Georgia,Onoe Tamehito,Mitobe Michihiro,Gao Jian-Dong,Okuhara Dayne,Tian Xin,Gallagher Anna-Rachel,Tang Zhangui,Xie Xiaoli,Lalioti Maria D.,Lee Ann-Hwee,Ehrlich Barbara E.,Somlo Stefan

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference82 articles.

1. Autosomal dominant polycystic kidney disease

2. A genetic interaction network of five genes for human polycystic kidney and liver diseases defines polycystin-1 as the central determinant of cyst formation

3. PKD1 interacts with PKD2 through a probable coiled-coil domain

4. Homo- and heterodimeric interactions between the gene products of PKD1 and PKD2

5. The Caenorhabditis elegans autosomal dominant polycystic kidney disease gene homologs lov-1 and pkd-2 act in the same pathway

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