Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic β cells recapitulates neonatal diabetes-Reference-Cited by-同舟云学术

Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic β cells recapitulates neonatal diabetes

Published:2008-12-08 Issue: Volume: Page:
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Girard Christophe A.,Wunderlich F. Thomas,Shimomura Kenju,Collins Stephan,Kaizik Stephan,Proks Peter,Abdulkader Fernando,Clark Anne,Ball Vicky,Zubcevic Lejla,Bentley Liz,Clark Rebecca,Church Chris,Hugill Alison,Galvanovskis Juris,Cox Roger,Rorsman Patrik,Brüning Jens C.,Ashcroft Frances M.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference37 articles.

1. Activating Mutations in Kir6.2 and Neonatal Diabetes

2. Mutations in KCNJ11, which encodes Kir6.2, are a common cause of diabetes diagnosed in the first 6 months of life, with the phenotype determined by genotype

3. ATP-sensitive K+ channels and disease: from molecule to malady

4. Switching from Insulin to Oral Sulfonylureas in Patients with Diabetes Due to Kir6.2 Mutations

5. Roles of K channels as metabolic sensors in acute metabolic changes

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