Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy-Reference-Cited by-同舟云学术

Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy

Published:2003-05-15 Issue:10 Volume:111 Page:1463-1474
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Yamamoto Shimako,Yang Guiping,Zablocki Daniela,Liu Jing,Hong Chull,Kim Song-Jung,Soler Sandra,Odashima Mari,Thaisz Jill,Yehia Ghassan,Molina Carlos A.,Yatani Atsuko,Vatner Dorothy E.,Vatner Stephen F.,Sadoshima Junichi

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference49 articles.

1. Apoptosis in Human Acute Myocardial Infarction

2. Hypoxia induces apoptosis with enhanced expression of Fas antigen messenger RNA in cultured neonatal rat cardiomyocytes.

3. Myocyte apoptosis during acute myocardial infarction in the mouse localizes to hypoxic regions but occurs independently of p53.

4. Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

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