TIGIT inhibition and lenalidomide synergistically promote antimyeloma immune responses after stem cell transplantation in mice
Author:
Funder
NIH
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/157907/files/pdf
Reference59 articles.
1. Bone marrow transplantation generates T cell–dependent control of myeloma in mice;Vuckovic;J Clin Invest,2019
2. Myeloma escape after stem cell transplantation is a consequence of T-cell exhaustion and is prevented by TIGIT blockade
3. TIGIT immune checkpoint blockade restores CD8+ T-cell immunity against multiple myeloma
4. T-cell Exhaustion in Multiple Myeloma Relapse after Autotransplant: Optimal Timing of Immunotherapy
5. T Cell Transcriptional Profiling and Immunophenotyping Uncover LAG3 as a Potential Significant Target of Immune Modulation in Multiple Myeloma
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1. Identification of HLA‐restricted Cytotoxic T Lymphocyte Epitopes of B Cell Maturation Antigen as Promising Peptide Vaccine Candidates for Preventive and Therapeutic Strategies in Multiple Myeloma;Advanced Therapeutics;2023-11-29
2. Definers and drivers of functional high-risk multiple myeloma: insights from genomic, transcriptomic, and immune profiling;Frontiers in Oncology;2023-10-02
3. High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma;Cell Reports Medicine;2023-10
4. Revisiting checkpoint inhibitors for myeloma: maintenance after stem cell transplant;Journal of Clinical Investigation;2023-02-15
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