Parkin is a lipid-responsive regulator of fat uptake in mice and mutant human cells-Reference-Cited by-同舟云学术

Parkin is a lipid-responsive regulator of fat uptake in mice and mutant human cells

Published:2011-09-01 Issue:9 Volume:121 Page:3701-3712
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Kim Kye-Young,Stevens Mark V.,Akter M. Hasina,Rusk Sarah E.,Huang Robert J.,Cohen Alexandra,Noguchi Audrey,Springer Danielle,Bocharov Alexander V.,Eggerman Tomas L.,Suen Der-Fen,Youle Richard J.,Amar Marcelo,Remaley Alan T.,Sack Michael N.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference47 articles.

1. Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

2. Parkin-deficient Mice Exhibit Nigrostriatal Deficits but Not Loss of Dopaminergic Neurons

3. Parkin-deficient mice are not a robust model of parkinsonism

4. Absence of nigral degeneration in aged parkin/DJ-1/PINK1 triple knockout mice

5. Structure of the Parkin in-between-ring domain provides insights for E3-ligase dysfunction in autosomal recessive Parkinson's disease

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