Liver-specific disruption of PPARγ in leptin-deficient mice improves fatty liver but aggravates diabetic phenotypes-Reference-Cited by-同舟云学术

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Liver-specific disruption of PPARγ in leptin-deficient mice improves fatty liver but aggravates diabetic phenotypes

Published:2003-03-01 Issue:5 Volume:111 Page:737-747
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Matsusue Kimihiko,Haluzik Martin,Lambert Gilles,Yim Sun-Hee,Gavrilova Oksana,Ward Jerrold M.,Brewer Bryan,Reitman Marc L.,Gonzalez Frank J.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference55 articles.

1. PPARs: transcriptional effectors of fatty acids and their derivatives

2. PPARs: Transcription Factors Controlling Lipid and Lipoprotein Metabolism

3. C/EBPα induces adipogenesis through PPARγ: a unified pathway

4. Conditional Disruption of the Peroxisome Proliferator-Activated Receptor γ Gene in Mice Results in Lowered Expression of ABCA1, ABCG1, and apoE in Macrophages and Reduced Cholesterol Efflux

5. A PPARγ-LXR-ABCA1 Pathway in Macrophages Is Involved in Cholesterol Efflux and Atherogenesis

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