MLK3 regulates bone development downstream of the faciogenital dysplasia protein FGD1 in mice-Reference-Cited by-同舟云学术

MLK3 regulates bone development downstream of the faciogenital dysplasia protein FGD1 in mice

Published:2011-11-01 Issue:11 Volume:121 Page:4383-4392
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Zou Weiguo,Greenblatt Matthew B.,Shim Jae-Hyuck,Kant Shashi,Zhai Bo,Lotinun Sutada,Brady Nicholas,Hu Dorothy Zhang,Gygi Steven P.,Baron Roland,Davis Roger J.,Jones Dallas,Glimcher Laurie H.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference45 articles.

1. A familial syndrome of short stature associated with facial dysplasia and genital anomalies

2. Isolation and characterization of the faciogenital dysplasia (Aarskog-Scott syndrome) gene: A putative RhoRac guanine nucleotide exchange factor

3. Faciogenital dysplasia protein (FGD1) and Vav, two related proteins required for normal embryonic development, are upstream regulators of Rho GTPases

4. The Faciogenital Dysplasia Gene Product FGD1 Functions as a Cdc42Hs-specific Guanine-Nucleotide Exchange Factor

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